Hair Loss & Scalp Vitality
Follicles need energy, circulation, and low oxidative stress to thicken and shine.
Questions to answer
When did you first notice hair thinning or changes in scalp health? Was there a clear trigger (illness, childbirth, stress, new med, diet change)?
Describe the pattern: diffuse thinning, receding hairline, patchy loss (alopecia areata), or increased shedding with preserved density?
How rapid is the change (weeks, months, years)? Any sudden increases in shedding?
Any scalp symptoms: itching, burning, flaking, redness, tenderness, or visible scarring?
Do you have recent lab results (CBC, ferritin, TSH, vitamin D, zinc, hormone panel)? If yes, paste values and dates.
What medications, supplements, or topical products are you using now (including minoxidil, finasteride, scalp steroids, retinoids)?
Any recent major life events or stressors, significant weight loss, changes in diet, or restrictive eating?
Do you have a family history of pattern hair loss, autoimmune conditions, or early thinning?
Describe your hair care routine: washing frequency, styling (heat, tight hairstyles), chemical treatments, hair density, and grooming habits.
Any history of skin conditions (psoriasis, seborrheic dermatitis, eczema) or scalp infections?
Do you have hormonal changes or reproductive history relevant to hair (pregnancy, postpartum, menopause, polycystic ovary syndrome)?
Any systemic symptoms: fatigue, menstrual irregularities, weight changes, or signs of androgen excess (acne, hirsutism)?
Have you tried interventions before? What helped or worsened (topicals, supplements, PRP, laser caps)?
What are your goals and timeline (regain density, reduce shedding, maintain current hair)?
Any red flags: rapidly progressive patches, painful swelling, pus, sudden total loss, or seizures/fever? (If yes, seek prompt clinical care.)
Bring these answers to our chat — they help Lorenzo tailor a safe, prioritized plan.
How scalp health, cellular energetics, inflammation, and light‑signaling relate to hair growth?
Hair growth depends on follicle cycling, local blood supply, hormonal signaling, and immune balance. Key mechanisms:
Follicle energetics and anagen maintenance: Hair follicles are metabolically active during the anagen (growth) phase. Mitochondrial ATP production supports cell division in the hair matrix; reduced cellular energetics can shorten anagen and increase shedding.
Microcirculation and nutrient delivery: Adequate capillary blood flow to follicles delivers oxygen, amino acids, and micronutrients critical for hair synthesis. Impaired circulation and microvascular inflammation correlate with thinning.
Hormonal and inflammatory modulation: Androgens (DHT) can miniaturize follicles in genetically susceptible individuals. Chronic inflammation, oxidative stress, and immune dysregulation (as in alopecia areata) also disrupt normal cycling.
Photobiomodulation and light‑signal approaches: Red and near‑infrared light have been shown to:
Stimulate mitochondrial activity (cytochrome c oxidase), increasing ATP and promoting follicle cell proliferation.
Enhance local microcirculation and reduce perifollicular inflammation.
Modulate local immune responses and oxidative stress, supporting recovery in inflammatory scalp conditions.
Adjunctive role: Light‑based therapies (low‑level laser/light therapy) and transdermal light‑signal patches are supportive tools that can improve shedding rates, increase hair density, and enhance hair thickness when used alongside targeted medical treatments (topical minoxidil, oral therapies, nutritional correction) and scalp care.
This is a gentle, non‑drug approach. It doesn’t force change; it invites your system to rebalance. Most people work in small daily steps and notice steady improvements.
Vera L.
Scalp felt healthier and my hair looks fuller in photos.
Ian F.
Less shedding and more confidence styling again.
Elena P.
Less shedding by week three and new baby hairs by week eight.
