
Cholesterol & Cellular Balance
Cholesterol is a transport system, not a villain. Balance the terrain and numbers often follow.
Questions to answer
When were you first told your cholesterol was high, or when did you first notice concerns? Any known trigger (weight change, new meds, diet shift, family diagnosis)?
Do you have a recent lipid panel? If yes, enter totals: LDL, HDL, triglycerides, and total cholesterol, and date of test.
Are you currently on cholesterol‑lowering medication (statins, ezetimibe, PCSK9 inhibitors) or supplements (fish oil, niacin, red yeast rice)? Please list names and doses.
Any family history of early cardiovascular disease or familial hypercholesterolemia (heart attack/stroke before age 55 in men or 65 in women)?
Do you have other metabolic or vascular conditions: diabetes, hypertension, obesity, fatty liver, or metabolic syndrome?
Describe your typical diet (mixed, low‑carb, Mediterranean, vegan), frequency of fried/processed foods, and weekly servings of fruits/vegetables.
How often do you exercise (type, intensity, minutes per session, days/week)? Include resistance training and aerobic activity.
Do you smoke, vape, or use nicotine products? How much and how long?
Any symptoms suggestive of cardiovascular disease: chest pain, shortness of breath on exertion, leg swelling or pain with walking, lightheadedness, or palpitations? (If yes, seek clinical evaluation.)
Any muscle pain, weakness, or unexplained fatigue (important if on statins)?
What is your current weight, height, and waist circumference (if known), and recent weight changes?
Any relevant labs in last 12 months (A1c, liver enzymes, kidney function, CRP)? If available, paste values and dates.
What lifestyle changes have you tried (diet, exercise, supplements)? What helped or didn’t and why?
What are your goals and time frame (e.g., reduce LDL by X, avoid medication, improve overall cardiovascular risk)?
Are you willing/able to follow a 30–90 day protocol (diet tweaks, activity changes, patch use, follow‑up labs)?
Bring these answers to our chat — it helps Lorenzo tailor your plan.
How cellular energetics, inflammation, and light‑based support relate to cholesterol
Cholesterol levels reflect complex interactions among hepatic lipid metabolism, systemic inflammation, cellular energy status, and lifestyle factors:
Hepatic synthesis and clearance: The liver produces cholesterol and clears LDL via LDL receptors; diet, genetics, and insulin resistance modulate these pathways.
Inflammation and oxidative stress: Chronic low‑grade inflammation and oxidative stress alter LDL particles (oxidized LDL), promote endothelial dysfunction, and accelerate atherosclerotic changes.
Cellular energy and metabolic signaling: Mitochondrial function affects lipid handling and insulin sensitivity; poor cellular energetics can worsen metabolic dysregulation.
Photobiomodulation and systemic effects: While light‑based interventions primarily target cellular energy (mitochondrial ATP production) and microcirculation, potential downstream effects could improve tissue metabolism and inflammation markers indirectly. These interventions are best viewed as adjunctive—supporting recovery, reducing oxidative stress, and improving circulation when combined with proven lipid‑lowering strategies.
Clinical context: Proven therapies to reduce cardiovascular risk include statins, diet (Mediterranean/DASH), weight loss, smoking cessation, blood pressure and glucose control, and physical activity. Light‑based patches or photobiomodulation may offer symptomatic support (energy, recovery, inflammation modulation) but should not replace guideline therapies for high cardiovascular risk.
This is a gentle, non‑drug approach. It doesn’t force change; it invites your system to rebalance. Most people work in small daily steps and notice steady improvements.




